JACC-Basic to Translational Science

Papers
(The H4-Index of JACC-Basic to Translational Science is 29. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-11-01 to 2024-11-01.)
ArticleCitations
Platelet and Endothelial Activation as Potential Mechanisms Behind the Thrombotic Complications of COVID-19 Patients151
SARS-CoV-2 Infects Human Engineered Heart Tissues and Models COVID-19 Myocarditis117
Decentralized Clinical Trials84
Targeting the Inflammasome in Cardiovascular Disease68
The Pathogenesis and Long-Term Consequences of COVID-19 Cardiac Injury65
Optimizing Foundational Therapies in Patients With HFrEF61
Iron Overload via Heme Degradation in the Endoplasmic Reticulum Triggers Ferroptosis in Myocardial Ischemia-Reperfusion Injury58
The Integrin Binding Peptide, ATN-161, as a Novel Therapy for SARS-CoV-2 Infection58
Intramyocardial Transplantation of Human iPS Cell–Derived Cardiac Spheroids Improves Cardiac Function in Heart Failure Animals57
Coronary Artery Disease Genetics Enlightened by Genome-Wide Association Studies57
Relationship of Nonalcoholic Fatty Liver Disease and Heart Failure With Preserved Ejection Fraction56
The Role of NLRP3 Inflammasome in Pericarditis52
Artificial Intelligence Discusses the Role of Artificial Intelligence in Translational Medicine51
Postacute Sequelae of Severe Acute Respiratory Syndrome Coronavirus 2 Infection50
Treatment Targets for Right Ventricular Dysfunction in Pulmonary Arterial Hypertension48
B Cells in Atherosclerosis46
In Situ Remodeling Overrules Bioinspired Scaffold Architecture of Supramolecular Elastomeric Tissue-Engineered Heart Valves41
Myopathic Cardiac Genotypes Increase Risk for Myocarditis41
Antisense Inhibition of Angiotensinogen With IONIS-AGT-LRx40
Bias in Medicine35
Galectin-3 Inhibition With Modified Citrus Pectin in Hypertension35
Neprilysin Inhibitors in Heart Failure35
TNFR1 Contributes to Activation-Induced Cell Death of Pathological CD4+ T Lymphocytes During Ischemic Heart Failure35
Selective FPR2 Agonism Promotes a Proresolution Macrophage Phenotype and Improves Cardiac Structure-Function Post Myocardial Infarction32
Nicotinamide Mononucleotide Alleviates Cardiomyopathy Phenotypes Caused by Short-Chain Enoyl-Coa Hydratase 1 Deficiency31
Selective Interleukin-6 Trans-Signaling Blockade Is More Effective Than Panantagonism in Reperfused Myocardial Infarction30
Interleukin-1β, Oxidative Stress, and Abnormal Calcium Handling Mediate Diabetic Arrhythmic Risk30
Fir(e)ing the Rhythm30
Cardiorenal Tissues Express SARS-CoV-2 Entry Genes and Basigin (BSG/CD147) Increases With Age in Endothelial Cells29
0.032173156738281