Molecular Metabolism

Papers
(The H4-Index of Molecular Metabolism is 47. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-03-01 to 2024-03-01.)
ArticleCitations
GLP-1 receptor agonists in the treatment of type 2 diabetes – state-of-the-art498
Monocarboxylate transporters in cancer327
Ketogenic diet in the treatment of cancer – Where do we stand?233
Metabolic dysfunction in polycystic ovary syndrome: Pathogenic role of androgen excess and potential therapeutic strategies208
Diabetes, infection risk and COVID-19200
Dysregulated lipid metabolism links NAFLD to cardiovascular disease190
Metabolic functions of the tumor suppressor p53: Implications in normal physiology, metabolic disorders, and cancer189
Metabolic-associated fatty liver disease and lipoprotein metabolism182
Compartmentalised acyl-CoA metabolism and roles in chromatin regulation142
Glucagon-like peptide-1 receptor co-agonists for treating metabolic disease141
A guide to understanding endoplasmic reticulum stress in metabolic disorders131
Nonalcoholic fatty liver disease (NAFLD) from pathogenesis to treatment concepts in humans130
Mechanisms of insulin resistance related to white, beige, and brown adipocytes124
Insulin action, type 2 diabetes, and branched-chain amino acids: A two-way street120
Formate metabolism in health and disease111
The melanocortin pathway and energy homeostasis: From discovery to obesity therapy110
GLP-1 physiology informs the pharmacotherapy of obesity109
Role of NAD+ in regulating cellular and metabolic signaling pathways102
Insulin action at a molecular level – 100 years of progress99
A non-proliferative role of pyrimidine metabolism in cancer98
Hepatic lipid droplets: A balancing act between energy storage and metabolic dysfunction in NAFLD96
Hepatokines and metabolism: Deciphering communication from the liver93
Metabolic drivers of non-alcoholic fatty liver disease90
Adapt and conquer: Metabolic flexibility in cancer growth, invasion and evasion90
Insulin-like growth factors: Ligands, binding proteins, and receptors87
The brain as an insulin-sensitive metabolic organ83
Imaging biomarkers of NAFLD, NASH, and fibrosis81
Phosphoenolpyruvate carboxykinase in cell metabolism: Roles and mechanisms beyond gluconeogenesis71
Genetic and epigenetic factors determining NAFLD risk68
Glucagon acutely regulates hepatic amino acid catabolism and the effect may be disturbed by steatosis66
Single-cell transcriptomics of human islet ontogeny defines the molecular basis of β-cell dedifferentiation in T2D63
Overexpression of the vitamin D receptor (VDR) induces skeletal muscle hypertrophy59
GLP-1 improves the supportive ability of astrocytes to neurons by promoting aerobic glycolysis in Alzheimer's disease58
Understanding insulin and its receptor from their three-dimensional structures57
Beta cell identity changes with mild hyperglycemia: Implications for function, growth, and vulnerability57
CD36 promotes de novo lipogenesis in hepatocytes through INSIG2-dependent SREBP1 processing56
Targeting the GIPR for obesity: To agonize or antagonize? Potential mechanisms55
Two genomes, one cell: Mitochondrial-nuclear coordination via epigenetic pathways55
Nicotinamide N-methyltransferase: At the crossroads between cellular metabolism and epigenetic regulation54
Ferroptosis and ferritinophagy in diabetes complications54
Sodium-glucose co-transporter2 expression and inflammatory activity in diabetic atherosclerotic plaques: Effects of sodium-glucose co-transporter2 inhibitor treatment54
Wnt/β-catenin signaling regulates adipose tissue lipogenesis and adipocyte-specific loss is rigorously defended by neighboring stromal-vascular cells52
Infusion of donor feces affects the gut–brain axis in humans with metabolic syndrome50
Irisin: Still chasing shadows50
Mitochondrial oxidative function in NAFLD: Friend or foe?50
Exploring the therapeutic potential of mitochondrial uncouplers in cancer49
Partial leptin deficiency confers resistance to diet-induced obesity in mice48
PPARγ and PPARα synergize to induce robust browning of white fat in vivo47
Targeting extracellular nutrient dependencies of cancer cells47
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