Acta Neuropathologica Communications

Papers
(The H4-Index of Acta Neuropathologica Communications is 36. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2021-02-01 to 2025-02-01.)
ArticleCitations
Decoding of the surfaceome and endocytome in primary glioblastoma cells identifies potential target antigens in the hypoxic tumor niche135
Dual-probe fluorescence spectroscopy for sensitive quantitation of Alzheimer’s amyloid pathology122
Immunological and tumor-intrinsic mechanisms mediate the synergistic growth suppression of experimental glioblastoma by radiotherapy and MET inhibition113
Axonal autophagic vesicle transport in the rat optic nerve in vivo under normal conditions and during acute axonal degeneration111
Dissection of transcriptomic and epigenetic heterogeneity of grade 4 gliomas: implications for prognosis108
Correction: Cleaved TMEM106B forms amyloid aggregates in central and peripheral nervous systems104
A novel case of glial transdifferentiation in renal medullary carcinoma brain metastasis100
The contribution of DNA methylation to the (dys)function of oligodendroglia in neurodegeneration95
Hyperspectral retinal imaging in Alzheimer’s disease and age-related macular degeneration: a review81
The inflammatory pathology of dysferlinopathy is distinct from calpainopathy, Becker muscular dystrophy, and inflammatory myopathies74
Asymmetric dysregulation of glutamate dynamics across the synaptic cleft in a mouse model of Alzheimer’s disease74
Astrocytes respond to a neurotoxic Aβ fragment with state-dependent Ca2+ alteration and multiphasic transmitter release62
A novel BRAF::PTPRN2 fusion in meningioma: a case report59
Spatially resolved transcriptomics reveals genes associated with the vulnerability of middle temporal gyrus in Alzheimer’s disease59
Potential prognostic determinants for FET::CREB fusion-positive intracranial mesenchymal tumor56
Comparison of transcriptome profiles between medulloblastoma primary and recurrent tumors uncovers novel variance effects in relapses53
Protein farnesylation is upregulated in Alzheimer’s human brains and neuron-specific suppression of farnesyltransferase mitigates pathogenic processes in Alzheimer’s model mice53
NRF2/ARE mediated antioxidant response to glaucoma: role of glia and retinal ganglion cells52
Prion strains associated with iatrogenic CJD in French and UK human growth hormone recipients51
Therapeutic targeting of Lyn kinase to treat chorea-acanthocytosis50
Multi-platform quantitation of alpha-synuclein human brain proteoforms suggests disease-specific biochemical profiles of synucleinopathies47
Focal amyloid and asymmetric tau in an imaging-to-autopsy case of clinical primary progressive aphasia with Alzheimer disease neuropathology47
Early biomarkers in the presymptomatic phase of cognitive impairment: changes in the endocannabinoidome and serotonergic pathways in Alzheimer's-prone mice after mTBI45
Two distinct conformers of PrPD type 1 of sporadic Creutzfeldt–Jakob disease with codon 129VV genotype faithfully propagate in vivo42
Oligodendrocyte lineage is severely affected in human alcohol-exposed foetuses41
The genomic alterations in glioblastoma influence the levels of CSF metabolites41
NAD salvage pathway machinery expression in normal and glaucomatous retina and optic nerve40
Pleomorphic xanthoastrocytoma in the multiverse of epigenomics: is it time to recognize the variants?39
Cortical microvascular raspberries and ageing: an independent but not exclusive relationship39
The neuropathological landscape of small vessel disease and Lewy pathology in a cohort of Hispanic and non-Hispanic White decedents with Alzheimer disease38
Deep histopathology genotype–phenotype analysis of focal cortical dysplasia type II differentiates between the GATOR1-altered autophagocytic subtype IIa and MTOR-altered migration deficient subtype II38
Clinicopathologic features of two unrelated autopsied patients with Charcot-Marie-Tooth disease carrying MFN2 gene mutation38
Systemic delivery of a specific antibody targeting the pathological N-terminal truncated tau peptide reduces retinal degeneration in a mouse model of Alzheimer’s Disease38
A pathogenic mutation in the ALS/FTD gene VCP induces mitochondrial hypermetabolism by modulating the permeability transition pore37
Alterations in iron content, iron-regulatory proteins and behaviour without tau pathology at one year following repetitive mild traumatic brain injury37
Pathological changes induced by Alzheimer’s brain inoculation in amyloid-beta plaque-bearing mice36
Development and characterization of novel anti-acetylated tau monoclonal antibodies to probe pathogenic tau species in Alzheimer’s disease36
Response to the letter by de Boer et al. (2022)36
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