Skeletal Muscle

Papers
(The TQCC of Skeletal Muscle is 9. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2022-08-01 to 2026-08-01.)
ArticleCitations
Enhancers integrate microenvironmental signals in muscle stem cells during regeneration in health, disease, and aging62
iPSC-derived skeletal muscle spheroids for Duchenne Muscular Dystrophy modeling41
Abnormalities in the genioglossus muscle and its neuromuscular synapse in leptin-deficient male mice33
Caloric restriction reprograms skeletal muscle molecular pathways in non-human primates: potential relevance to human aging biology31
The paradox of hnRNPK: both absence and excess impair skeletal muscle function in mice30
Tropomyosin 3 (TPM3) function in skeletal muscle and in myopathy28
Metabolic signatures and potential biomarkers of sarcopenia in suburb-dwelling older Chinese: based on untargeted GC–MS and LC–MS26
Non-regenerative myogenesis in adult skeletal muscles: myofiber death-independent muscle satellite cell expansion25
Targeting muscle fibrosis using non-specific collagenase injections destabilizes the basal lamina and induces matrix remodeling22
Pirfenidone treatment improves ischemic muscle function in mice with chronic kidney disease20
Golodirsen restores DMD transcript imbalance in Duchenne Muscular Dystrophy patient muscle cells19
Pilot investigations into the mechanistic basis for adverse effects of glucocorticoids in dysferlinopathy18
Zebrafish and cellular models of SELENON-Congenital myopathy exhibit novel embryonic and metabolic phenotypes17
A knock down strategy for rapid, generic, and versatile modelling of muscular dystrophies in 3D-tissue-engineered-skeletal muscle16
ASM is a therapeutic target in dermatomyositis by regulating the differentiation of naive CD4 + T cells into Th17 and Treg subsets16
SIX transcription factors are necessary for the activation of DUX4 expression in facioscapulohumeral muscular dystrophy15
Fibro-adipogenic progenitor cells in skeletal muscle unloading: metabolic and functional impairments15
Duchenne muscular dystrophy: from gene to gene-ius therapies15
Comparative lipidomic and metabolomic profiling of mdx and severe mdx-apolipoprotein e-null mice14
Spiny mice are primed but fail to regenerate volumetric skeletal muscle loss injuries13
AAV-NRIP gene therapy ameliorates motor neuron degeneration and muscle atrophy in ALS model mice13
Integrin αVβ5 regulates myoblast proliferation and differentiation in sarcopenia mice treated with FNDC5 gene delivery12
Extensive striated muscle damage in a rat model of Duchenne muscular dystrophy with Dmd exons 10–17 duplication12
New tools for the investigation of muscle fiber-type spatial distributions across histological sections12
Functional and structural pathologies in skeletal muscle of a rat model of Duchenne muscular dystrophy11
The contribution of skeletal muscle interstitial cells to myogenesis11
Pannexin 1 dysregulation in Duchenne muscular dystrophy and its exacerbation of dystrophic features in mdx mice11
Necessity of Notch3 signaling in myofiber maturation in a pluripotent stem cell transplant model11
Expression of Myomaker and Myomerger in myofibers causes muscle pathology11
Elizabeth Chen is elected to the U.S. National Academy of Sciences11
Generation of a novel mouse model of nemaline myopathy due to recurrent NEB exon 55 deletion10
Decreased number of satellite cells-derived myonuclei in both fast- and slow-twitch muscles in HeyL-KO mice during voluntary running exercise10
A 3D skeletal muscle system for disease modelling and secretome profiling of Duchenne muscular dystrophy9
Eldecalcitol prevents muscle loss and osteoporosis in disuse muscle atrophy via NF-κB signaling in mice9
Fusion of myofibre branches is a physiological feature of healthy human skeletal muscle regeneration9
Macroglossia and less advanced dystrophic change in the tongue muscle of the Duchenne muscular dystrophy rat9
Fighting for every beat: cardiac therapies in Duchenne muscular dystrophy9
Endurance exercise with reduced muscle glycogen content influences substrate utilization and attenuates acute mTORC1- and autophagic signaling in human type I and type II muscle fibers9
Exercise, disease state and sex influence the beneficial effects of Fn14-depletion on survival and muscle pathology in the SOD1G93A amyotrophic lateral sclerosis (ALS) mouse model9
Wnt7a is required for regeneration of dystrophic skeletal muscle9
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