CNS Neuroscience & Therapeutics

Papers
(The H4-Index of CNS Neuroscience & Therapeutics is 34. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-11-01 to 2024-11-01.)
ArticleCitations
Peripheral inflammation and blood–brain barrier disruption: effects and mechanisms300
Zinc attenuates ferroptosis and promotes functional recovery in contusion spinal cord injury by activating Nrf2/GPX4 defense pathway143
Microglial/Macrophage polarization and function in brain injury and repair after stroke106
Neurovascular Unit: A critical role in ischemic stroke105
Quality of life in Parkinson's disease: A systematic review and meta‐analysis of comparative studies97
Pathogenesis and pathophysiology of idiopathic normal pressure hydrocephalus81
Gut bacterial profiles in Parkinson's disease: A systematic review77
Phagocytic microglia and macrophages in brain injury and repair74
Acyl‐CoA synthetase long chain family member 4 plays detrimental role in early brain injury after subarachnoid hemorrhage in rats by inducing ferroptosis62
Perioperative neurocognitive disorders: A narrative review focusing on diagnosis, prevention, and treatment60
Ferroptosis‐related gene signature predicts prognosis and immunotherapy in glioma58
Fisetin ameliorates cognitive impairment by activating mitophagy and suppressing neuroinflammation in rats with sepsis‐associated encephalopathy53
Kynurenic acid in neurodegenerative disorders—unique neuroprotection or double‐edged sword?53
Neuroprotective effects and mechanisms of ischemic/hypoxic preconditioning on neurological diseases51
Ketogenic diet ameliorates cognitive impairment and neuroinflammation in a mouse model of Alzheimer’s disease50
Ferroptosis contributes to hypoxic–ischemic brain injury in neonatal rats: Role of the SIRT1/Nrf2/GPx4 signaling pathway49
Taurine and oxidative stress in retinal health and disease48
Electroacupuncture ameliorates postoperative cognitive dysfunction and associated neuroinflammation via NLRP3 signal inhibition in aged mice48
Endothelial glycocalyx as an important factor in composition of blood‐brain barrier47
Brain Energy Metabolism: Astrocytes in Neurodegenerative Diseases46
Liraglutide attenuate central nervous inflammation and demyelination through AMPK and pyroptosis‐related NLRP3 pathway45
Srs11‐92, a ferrostatin‐1 analog, improves oxidative stress and neuroinflammation via Nrf2 signal following cerebral ischemia/reperfusion injury44
Animal models for the study of depressive disorder44
Brain stimulation: a therapeutic approach for the treatment of neurological disorders43
Regulatory T cells in ischemic stroke43
Recent advances and perspectives of postoperative neurological disorders in the elderly surgical patients42
Central nervous system diseases related to pathological microglial phagocytosis40
AIM2 deletion enhances blood‐brain barrier integrity in experimental ischemic stroke40
Intranasal insulin improves mitochondrial function and attenuates motor deficits in a rat 6‐OHDA model of Parkinson's disease38
Stress hyperglycemia is associated with in‐hospital mortality in patients with diabetes and acute ischemic stroke38
Lipocalin 2 regulates iron homeostasis, neuroinflammation, and insulin resistance in the brains of patients with dementia: Evidence from the current literature37
Morinda officinalis oligosaccharides alleviate depressive‐like behaviors in post‐stroke rats via suppressing NLRP3 inflammasome to inhibit hippocampal inflammation36
Demyelinating processes in aging and stroke in the central nervous system and the prospect of treatment strategy36
SerpinA3N attenuates ischemic stroke injury by reducing apoptosis and neuroinflammation36
Fine particulate matter exposure aggravates ischemic injury via NLRP3 inflammasome activation and pyroptosis34
Gut dysbiosis in stroke and its implications on Alzheimer’s disease‐like cognitive dysfunction34
Sevoflurane preconditioning protects experimental ischemic stroke by enhancing anti‐inflammatory microglia/macrophages phenotype polarization through GSK‐3β/Nrf2 pathway34
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