Frontiers in Molecular Neuroscience

Papers
(The H4-Index of Frontiers in Molecular Neuroscience is 36. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-11-01 to 2024-11-01.)
ArticleCitations
The Neuromuscular Junction in Health and Disease: Molecular Mechanisms Governing Synaptic Formation and Homeostasis105
The Role of Neurod Genes in Brain Development, Function, and Disease92
SOX Transcription Factors as Important Regulators of Neuronal and Glial Differentiation During Nervous System Development and Adult Neurogenesis87
New Insights Into Drug Discovery Targeting Tau Protein87
Mouse Models of Alzheimer’s Disease83
Neuronal cell death mechanisms in Alzheimer’s disease: An insight73
Insights Into the Role and Potential of Schwann Cells for Peripheral Nerve Repair From Studies of Development and Injury69
Recent Developments in Data Independent Acquisition (DIA) Mass Spectrometry: Application of Quantitative Analysis of the Brain Proteome63
Mitochondrial Extracellular Vesicles – Origins and Roles61
Long Non-coding RNAs and Circular RNAs: Insights Into Microglia and Astrocyte Mediated Neurological Diseases61
New insight into neurological degeneration: Inflammatory cytokines and blood–brain barrier59
Current Approaches for Glioma Gene Therapy and Virotherapy58
Microglial Exosomes in Neurodegenerative Disease55
A stay of execution: ATF4 regulation and potential outcomes for the integrated stress response52
Ketone Bodies in the Brain Beyond Fuel Metabolism: From Excitability to Gene Expression and Cell Signaling50
Glycogen Synthase Kinase-3 Inhibitors: Preclinical and Clinical Focus on CNS-A Decade Onward49
More than a marker: potential pathogenic functions of MAP249
Automatic autism spectrum disorder detection using artificial intelligence methods with MRI neuroimaging: A review48
Opioid Receptor-Mediated Regulation of Neurotransmission in the Brain47
Current and Future Prospects for Gene Therapy for Rare Genetic Diseases Affecting the Brain and Spinal Cord46
Modulation of β-Amyloid Fibril Formation in Alzheimer’s Disease by Microglia and Infection45
The Structure and Function of Ionotropic Receptors in Drosophila43
Zebrafish as a model organism for neurodegenerative disease42
Wnt/β-Catenin-Dependent Transcription in Autism Spectrum Disorders41
Hyperactivity Induced by Soluble Amyloid-β Oligomers in the Early Stages of Alzheimer's Disease41
Using Zebrafish to Model Autism Spectrum Disorder: A Comparison of ASD Risk Genes Between Zebrafish and Their Mammalian Counterparts39
SNARE Regulatory Proteins in Synaptic Vesicle Fusion and Recycling38
Lipid Metabolism Influence on Neurodegenerative Disease Progression: Is the Vehicle as Important as the Cargo?38
A Neurotoxic Ménage-à-trois: Glutamate, Calcium, and Zinc in the Excitotoxic Cascade38
Amyloid-β Peptide Impact on Synaptic Function and Neuroepigenetic Gene Control Reveal New Therapeutic Strategies for Alzheimer’s Disease38
Effects of Mdivi-1 on Neural Mitochondrial Dysfunction and Mitochondria-Mediated Apoptosis in Ischemia-Reperfusion Injury After Stroke: A Systematic Review of Preclinical Studies38
The Unfolded Protein Responses in Health, Aging, and Neurodegeneration: Recent Advances and Future Considerations37
Astrocytic and Oligodendrocytic P2X7 Receptors Determine Neuronal Functions in the CNS37
Neuroinflammation in Ischemic Stroke: Focus on MicroRNA-mediated Polarization of Microglia37
MicroRNA Dysregulation in Epilepsy: From Pathogenetic Involvement to Diagnostic Biomarker and Therapeutic Agent Development37
AAV Targeting of Glial Cell Types in the Central and Peripheral Nervous System and Relevance to Human Gene Therapy37
Overloaded Adeno-Associated Virus as a Novel Gene Therapeutic Tool for Otoferlin-Related Deafness36
Neuroligin-3: A Circuit-Specific Synapse Organizer That Shapes Normal Function and Autism Spectrum Disorder-Associated Dysfunction36
Phosphorylation and O-GlcNAcylation of the PHF-1 Epitope of Tau Protein Induce Local Conformational Changes of the C-Terminus and Modulate Tau Self-Assembly Into Fibrillar Aggregates36
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