Traffic

Papers
(The TQCC of Traffic is 6. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2022-06-01 to 2026-06-01.)
ArticleCitations
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Diamond controls epithelial polarity through the dynactin‐dynein complex20
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AP2S1 regulates APP degradation through late endosome–lysosome fusion in cells and APP/PS1 mice16
Role of trafficking protein particle complex 2 in medaka development15
SWIP mediates retromer‐independent membrane recruitment of the WASH complex15
Beyond the Secretory Pathway: New Insights Into Protein Release15
Clathrin‐Mediated Endocytosis in Plants: Historical to Modern Advances14
Acute GARP Depletion Disrupts Vesicle Transport, Leading to Severe Defects in Sorting, Secretion and O 13
Dynamics of axonal β‐actin mRNA in live hippocampal neurons13
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Restoration of β‐GC trafficking improves the lysosome function in Gaucher disease12
Adaptor‐Mediated Trafficking of Tank Binding Kinase 1 During Diverse Cellular Processes12
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Copper Homeostasis at the Crossroads of Cellular Metabolism, Epigenetic Regulation and Protein Trafficking12
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Rescue of secretion of rare‐disease‐associated misfolded mutant glycoproteins in UGGT1 knock‐out m11
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Clathrins Are Involved in the Endocytosis of Host Cytosol in the Malaria Parasite10
α‐Synuclein fibrils explore actin‐mediated macropinocytosis for cellular entry into model neuroblastoma neurons10
N‐BAR and F‐BAR proteins—endophilin‐A3 and PSTPIP1—control clathrin‐independent endocytosis of L1CAM10
Peroxisome population control by phosphoinositide signaling at the endoplasmic reticulum‐plasma membrane interface10
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TENT5/FAM46: An Enigmatic Family of Secretory Tuners9
Myosin V facilitates polarised E‐cadherin secretion9
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Endoglin mutants retained in the endoplasmic reticulum exacerbate loss of function in hereditary hemorrhagic telangiectasia type 1 (HHT1) by exerting dominant negative effects on the wild type allele8
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Cholangiocytes express an isoform of soluble adenylyl cyclase that is N‐linked glycosylated and secreted in extracellular vesicles8
Acute COG complex inactivation unveiled its immediate impact on Golgi and illuminated the nature of intra‐Golgi recycling vesicles7
Molecular mechanisms of PI4K regulation and their involvement in viral replication7
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Inhibition of Autophagy Alters Intracellular Transport of  APP Resulting in Increased APP Processi7
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GTP‐stimulated membrane fission by the N‐BAR protein AMPH‐16
Intercellular Mitochondrial Transfer: The Novel Therapeutic Mechanism for Diseases6
Disease‐Associated Factors at the Endoplasmic Reticulum–Golgi Interface6
Inhibition of cholesterol transport impairs Cav‐1 trafficking and small extracellular vesicles secretion, promoting amphisome formation in melanoma cells6
EFA6A , an Exchange Factor for Arf6, Regulates NGF ‐Dependent TrkA 6
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