Basic Research in Cardiology

Papers
(The TQCC of Basic Research in Cardiology is 27. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-10-01 to 2024-10-01.)
ArticleCitations
Resolving the intertwining of inflammation and fibrosis in human heart failure at single-cell level107
Small extracellular vesicles obtained from hypoxic mesenchymal stromal cells have unique characteristics that promote cerebral angiogenesis, brain remodeling and neurological recovery after focal cere96
Reduced reticulum–mitochondria Ca2+ transfer is an early and reversible trigger of mitochondrial dysfunctions in diabetic cardiomyopathy86
IMproving Preclinical Assessment of Cardioprotective Therapies (IMPACT) criteria: guidelines of the EU-CARDIOPROTECTION COST Action86
Cellular and mitochondrial mechanisms of atrial fibrillation77
PCSK9 regulates pyroptosis via mtDNA damage in chronic myocardial ischemia67
Coronary blood flow in heart failure: cause, consequence and bystander61
Mitochondrial calpain-1 activates NLRP3 inflammasome by cleaving ATP5A1 and inducing mitochondrial ROS in CVB3-induced myocarditis58
Mouse models of atherosclerosis and their suitability for the study of myocardial infarction58
Cytokines as therapeutic targets for cardio- and cerebrovascular diseases58
Protective effect of HINT2 on mitochondrial function via repressing MCU complex activation attenuates cardiac microvascular ischemia–reperfusion injury54
Blunting TRPML1 channels protects myocardial ischemia/reperfusion injury by restoring impaired cardiomyocyte autophagy48
Does remote ischaemic conditioning reduce inflammation? A focus on innate immunity and cytokine response47
Hyperglycemia regulates cardiac K+ channels via O-GlcNAc-CaMKII and NOX2-ROS-PKC pathways47
Regulation of STAT3 and its role in cardioprotection by conditioning: focus on non-genomic roles targeting mitochondrial function46
Cardiomyocyte Na+ and Ca2+ mishandling drives vicious cycle involving CaMKII, ROS, and ryanodine receptors41
Plasminogen activator inhibitor-1 reduces cardiac fibrosis and promotes M2 macrophage polarization in inflammatory cardiomyopathy41
Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering39
Fibroblast GATA-4 and GATA-6 promote myocardial adaptation to pressure overload by enhancing cardiac angiogenesis38
Increased susceptibility of human endothelial cells to infections by SARS-CoV-2 variants37
Cardioprotection by selective SGLT-2 inhibitors in a non-diabetic mouse model of myocardial ischemia/reperfusion injury: a class or a drug effect?37
miR-106a–363 cluster in extracellular vesicles promotes endogenous myocardial repair via Notch3 pathway in ischemic heart injury37
Cardioprotection by remote ischemic conditioning is transferable by plasma and mediated by extracellular vesicles36
Glucagon-like peptide-1 (GLP-1) receptor activation dilates cerebral arterioles, increases cerebral blood flow, and mediates remote (pre)conditioning neuroprotection against ischaemic stroke36
Resident cardiac macrophages: crucial modulators of cardiac (patho)physiology35
Adenosine and adenosine receptor-mediated action in coronary microcirculation35
Targeting myocardial ischaemic injury in the absence of reperfusion34
LncRNA LncHrt preserves cardiac metabolic homeostasis and heart function by modulating the LKB1-AMPK signaling pathway31
Macrophage depletion in stellate ganglia alleviates cardiac sympathetic overactivation and ventricular arrhythmogenesis by attenuating neuroinflammation in heart failure30
Connexin 43 phosphorylation by casein kinase 1 is essential for the cardioprotection by ischemic preconditioning30
Regulatory B cells improve ventricular remodeling after myocardial infarction by modulating monocyte migration29
SAIL: a new conserved anti-fibrotic lncRNA in the heart29
AMPKα1 deletion in myofibroblasts exacerbates post-myocardial infarction fibrosis by a connexin 43 mechanism28
Thiol-based redox-active proteins as cardioprotective therapeutic agents in cardiovascular diseases28
Functional investigation of the coronary artery disease gene SVEP128
Transcriptome-wide association study of coronary artery disease identifies novel susceptibility genes27
Reverse re-modelling chronic heart failure by reinstating heart rate variability27
HIMF deletion ameliorates acute myocardial ischemic injury by promoting macrophage transformation to reparative subtype27
Remote ischaemic conditioning: defining critical criteria for success—report from the 11th Hatter Cardiovascular Workshop27
The zinc transporter ZIP7 (Slc39a7) controls myocardial reperfusion injury by regulating mitophagy27
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