Basic Research in Cardiology

Papers
(The TQCC of Basic Research in Cardiology is 28. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2021-12-01 to 2025-12-01.)
ArticleCitations
Correction to: Endothelial actions of atrial natriuretic peptide prevent pulmonary hypertension in mice108
Endothelial ACKR3 drives atherosclerosis by promoting immune cell adhesion to vascular endothelium101
Erythrocytes from patients with ST-elevation myocardial infarction induce cardioprotection through the purinergic P2Y13 receptor and nitric oxide signaling81
Elevated platelet–leukocyte complexes are associated with, but dispensable for myocardial ischemia–reperfusion injury81
Alkaline nucleoplasm facilitates contractile gene expression in the mammalian heart51
Spinal cord astrocytes regulate myocardial ischemia–reperfusion injury48
Development and characterization of anti-fibrotic natural compound similars with improved effectivity40
Tachycardiomyopathy entails a dysfunctional pattern of interrelated mitochondrial functions40
Publisher Correction: Transcriptome-wide association study of coronary artery disease identifies novel susceptibility genes38
New insights into cardioprotection, gained by adopting the CAESAR standards of rigor35
Desmin intermediate filaments and tubulin detyrosination stabilize growing microtubules in the cardiomyocyte34
Retraction Note to: Amphiregulin enhances cardiac fibrosis and aggravates cardiac dysfunction in mice with experimental myocardial infarction partly through activating EGFR‑dependent pathway34
On the cellular origin of cardiosphere-derived cells (CDCs)31
Reverse re-modelling chronic heart failure by reinstating heart rate variability30
Uncovering the molecular identity of cardiosphere-derived cells (CDCs) by single-cell RNA sequencing30
Mechanism of the switch from NO to H2O2 in endothelium-dependent vasodilation in diabetes29
Increased protein S-nitrosylation in mitochondria: a key mechanism of exercise-induced cardioprotection28
Fibro-fatty remodelling in arrhythmogenic cardiomyopathy28
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