OOIR: Observatory of International Research

Papers

(The TQCC of Basic Research in Cardiology is 26. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-07-01 to 2024-07-01.)

Article	Citations
Resolving the intertwining of inflammation and fibrosis in human heart failure at single-cell level	101
Small extracellular vesicles obtained from hypoxic mesenchymal stromal cells have unique characteristics that promote cerebral angiogenesis, brain remodeling and neurological recovery after focal cere	85
IMproving Preclinical Assessment of Cardioprotective Therapies (IMPACT) criteria: guidelines of the EU-CARDIOPROTECTION COST Action	83
Reduced reticulum–mitochondria Ca2+ transfer is an early and reversible trigger of mitochondrial dysfunctions in diabetic cardiomyopathy	78
Cellular and mitochondrial mechanisms of atrial fibrillation	68
PCSK9 regulates pyroptosis via mtDNA damage in chronic myocardial ischemia	61
Coronary blood flow in heart failure: cause, consequence and bystander	57
Non-coding RNAs: emerging players in cardiomyocyte proliferation and cardiac regeneration	55
Mouse models of atherosclerosis and their suitability for the study of myocardial infarction	53
Cytokines as therapeutic targets for cardio- and cerebrovascular diseases	52
Protective effect of HINT2 on mitochondrial function via repressing MCU complex activation attenuates cardiac microvascular ischemia–reperfusion injury	50
Mitochondrial calpain-1 activates NLRP3 inflammasome by cleaving ATP5A1 and inducing mitochondrial ROS in CVB3-induced myocarditis	48
Hyperglycemia regulates cardiac K+ channels via O-GlcNAc-CaMKII and NOX2-ROS-PKC pathways	47
Regulation of STAT3 and its role in cardioprotection by conditioning: focus on non-genomic roles targeting mitochondrial function	42
Does remote ischaemic conditioning reduce inflammation? A focus on innate immunity and cytokine response	42
Blunting TRPML1 channels protects myocardial ischemia/reperfusion injury by restoring impaired cardiomyocyte autophagy	42
Neuraminidase-1 promotes heart failure after ischemia/reperfusion injury by affecting cardiomyocytes and invading monocytes/macrophages	41
Inhibition of macrophage proliferation dominates plaque regression in response to cholesterol lowering	40
NAD+ administration decreases microvascular damage following cardiac ischemia/reperfusion by restoring autophagic flux	39
Fibroblast GATA-4 and GATA-6 promote myocardial adaptation to pressure overload by enhancing cardiac angiogenesis	37
Cardiomyocyte Na+ and Ca2+ mishandling drives vicious cycle involving CaMKII, ROS, and ryanodine receptors	36
Plasminogen activator inhibitor-1 reduces cardiac fibrosis and promotes M2 macrophage polarization in inflammatory cardiomyopathy	36
miR-106a–363 cluster in extracellular vesicles promotes endogenous myocardial repair via Notch3 pathway in ischemic heart injury	36
Increased susceptibility of human endothelial cells to infections by SARS-CoV-2 variants	34
Resident cardiac macrophages: crucial modulators of cardiac (patho)physiology	33

Targeting myocardial ischaemic injury in the absence of reperfusion	33
Metoprolol blunts the time-dependent progression of infarct size	33
Glucagon-like peptide-1 (GLP-1) receptor activation dilates cerebral arterioles, increases cerebral blood flow, and mediates remote (pre)conditioning neuroprotection against ischaemic stroke	33
Cardioprotection by remote ischemic conditioning is transferable by plasma and mediated by extracellular vesicles	32
Adenosine and adenosine receptor-mediated action in coronary microcirculation	32
Macrophage depletion in stellate ganglia alleviates cardiac sympathetic overactivation and ventricular arrhythmogenesis by attenuating neuroinflammation in heart failure	29
Cardioprotection by selective SGLT-2 inhibitors in a non-diabetic mouse model of myocardial ischemia/reperfusion injury: a class or a drug effect?	28
SAIL: a new conserved anti-fibrotic lncRNA in the heart	28
Macrophage-mediated regulation of catecholamines in sympathetic neural remodeling after myocardial infarction	28
Selective NADH communication from α-ketoglutarate dehydrogenase to mitochondrial transhydrogenase prevents reactive oxygen species formation under reducing conditions in the heart	27
Functional investigation of the coronary artery disease gene SVEP1	27
Connexin 43 phosphorylation by casein kinase 1 is essential for the cardioprotection by ischemic preconditioning	27
LncRNA LncHrt preserves cardiac metabolic homeostasis and heart function by modulating the LKB1-AMPK signaling pathway	27
AMPKα1 deletion in myofibroblasts exacerbates post-myocardial infarction fibrosis by a connexin 43 mechanism	26
Interruption of vascular endothelial growth factor receptor 2 signaling induces a proliferative pulmonary vasculopathy and pulmonary hypertension	26
The zinc transporter ZIP7 (Slc39a7) controls myocardial reperfusion injury by regulating mitophagy	26
HIMF deletion ameliorates acute myocardial ischemic injury by promoting macrophage transformation to reparative subtype	26