Molecular Pharmacology

Papers
(The H4-Index of Molecular Pharmacology is 17. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2022-05-01 to 2026-05-01.)
ArticleCitations
A 19F-qNMR-Guided Mathematical Model for G Protein-Coupled Receptor Signaling69
Simplified Method for Kinetic and Thermodynamic Screening of Cardiotonic Steroids through the K+-Dependent Phosphatase Activity of Na+/K+-ATPase with Chromogenic pNPP Substrate44
Fat traffic control: S-acylation in axonal transport37
In Vitro Pharmacological Profile of KW-6356, a Novel Adenosine A2A Receptor Antagonist/Inverse Agonist37
Enhancing remyelination in multiple sclerosis via M1 muscarinic acetylcholine receptor36
UCM-A86 is a selective positive allosteric modulator of GluN1/GluN3 NMDA receptors35
Structural Basis for the Allosteric Inhibition of Hypoxia-Inducible Factor 2 by Belzutifan32
Expression of concern: “Adelmidrol, a Palmitoylethanolamide Analogue, as a New PharmacologicalTreatment for the Management of Inflammatory Bowel Disease” [Mol. Pharmacol. 2016; 90: 549-561] by Cordaro29
Editorial Board28
Editorial Board28
Table of Contents28
Mechanism-based inactivation of human aldehyde oxidase by erlotinib: Mechanistic insights from structural analogs and molecular docking24
Thermogenic Modulation of Adipose Depots: A Perspective on Possible Therapeutic Intervention with Early Cardiorenal Complications of Metabolic Impairment20
Development of a Novel Assay for Direct Assessment of Selective Amylin Receptor Activation Reveals Novel Differences in Behavior of Selective and Nonselective Peptide Agonists18
Regulation of organic anion transporting polypeptide 1B1 transport function by concurrent phosphorylation and lysine-acetylation: A novel posttranslational regulation mechanism17
Cholesterol residence time drives regulation of the G protein-coupled cholecystokinin receptor17
Isoprenaline shows unique kinase dependencies in stimulating β1AR–β-arrestin2 interaction compared to endogenous catecholamines17
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