Journal of Neuropathology and Experimental Neurology

Papers
(The H4-Index of Journal of Neuropathology and Experimental Neurology is 17. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2020-11-01 to 2024-11-01.)
ArticleCitations
The Second NINDS/NIBIB Consensus Meeting to Define Neuropathological Criteria for the Diagnosis of Chronic Traumatic Encephalopathy122
Early Selective Vulnerability of the CA2 Hippocampal Subfield in Primary Age-Related Tauopathy40
Fundamentals of the Development of Connectivity in the Human Fetal Brain in Late Gestation: From 24 Weeks Gestational Age to Term37
Advances in Deep Neuropathological Phenotyping of Alzheimer Disease: Past, Present, and Future31
Multiple Sclerosis: Microglia, Monocytes, and Macrophage-Mediated Demyelination28
Deep Learning-Based Image Classification in Differentiating Tufted Astrocytes, Astrocytic Plaques, and Neuritic Plaques27
Chronic Traumatic Encephalopathy (CTE)-Type Neuropathology in a Young Victim of Domestic Abuse27
SARS-CoV-2 Brain Regional Detection, Histopathology, Gene Expression, and Immunomodulatory Changes in Decedents with COVID-1926
LATE Neuropathologic Changes with Little or No Alzheimer Disease is Common and is Associated with Cognitive Impairment but Not Frontotemporal Dementia23
Hemodynamic Force as a Potential Regulator of Inflammation-Mediated Focal Growth of Saccular Aneurysms in a Rat Model21
Rat and Mouse Brain Tumor Models for Experimental Neuro-Oncology Research21
Detailed Clinical and Histopathological Description of 8 Cases of Molecularly Defined CNS Neuroblastomas20
Asymmetry of Hippocampal Tau Pathology in Primary Age-Related Tauopathy and Alzheimer Disease20
Expression of GFAP and Tau Following Blast Exposure in the Cerebral Cortex of Ferrets18
Vascular Lesions, APOE ε4, and Tau Pathology in Alzheimer Disease18
Polymorphous Low-Grade Neuroepithelial Tumor of the Young (PLNTY): Molecular Profiling Confirms Frequent MAPK Pathway Activation17
Tau Pathology in Chronic Traumatic Encephalopathy is Primarily Neuronal17
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