Journal of Cell Biology

Papers
(The H4-Index of Journal of Cell Biology is 45. The table below lists those papers that are above that threshold based on CrossRef citation counts [max. 250 papers]. The publications cover those that have been published in the past four years, i.e., from 2022-05-01 to 2026-05-01.)
ArticleCitations
Sugar-free synapses run on mitochondrial Sirtuin 3426
Distinct roles of two homologous kinesins in mammalian motile cilia159
A function of TPL/TBL1-type corepressors is to nucleate the assembly of the preinitiation complex125
Diverse microtubule-binding repeats regulate TPX2 activities at distinct locations within the spindle107
PI(3)P regulates mitochondrial dynamics through FGD-dependent actin organization106
Stress granules and mTOR are regulated by membrane atg8ylation during lysosomal damage97
A conserved role for centriolar satellites in translation of centrosomal and ciliary proteins96
Lipid droplets: Open questions and conceptual advances around a unique organelle95
A collective strategy to promote the dissemination of single cancer cells94
GARLH regulates neuroligin preference for excitatory versus inhibitory synapses91
Cholesterol depletion activates trafficking-coupled sphingolipid synthesis88
Conformational transitions of the Spindly adaptor underlie its interaction with Dynein and Dynactin86
GxcM-Fbp17/RacC-WASP signaling regulates polarized cortex assembly in migrating cells via Arp2/386
Developmental pruning of sensory neurites by mechanical tearing in Drosophila84
Kinetochores grip microtubules with directionally asymmetric strength79
DBF4, not DRF1, is the crucial regulator of CDC7 kinase at replication forks76
VPS13B is localized at the interface between Golgi cisternae and is a functional partner of FAM177A175
Homocysteine disrupts lysosomal function by V-ATPase inhibition74
Distinct TRAPP complexes activate Ypt/Rab GTPases in secretion and autophagy71
RIM and MUNC13 membrane–binding domains are essential for neuropeptide secretion71
Microtubule-driven cell shape changes and actomyosin flow synergize to position the centrosome64
Edge-vertex flow enables rapid adhesion reinforcement under tension63
Tld1 is a regulator of triglyceride lipolysis that demarcates a lipid droplet subpopulation60
Reticulons promote formation of ER-derived double-membrane vesicles that facilitate SARS-CoV-2 replication59
ESCRT-I and PTPN23 mediate microautophagy of ubiquitylated tau aggregates59
PP6 regulation of Aurora A–TPX2 limits NDC80 phosphorylation and mitotic spindle size58
Mechanisms of genome instability from cellular folate stress56
The α-Catenin mechanosensing M region is required for cell adhesion during tissue morphogenesis55
The PAX3-FOXO1 fusion gene reduces cell–ECM interactions and TGFβ signaling in rhabdomyosarcoma55
Rab GTPases as “eat me” signals for selective autophagy54
Bill Weis (1959–2023): Pioneering structural biologist and biochemist who revolutionized our understanding of cell adhesion and Wnt signaling53
TIMP-1 is a novel ligand of Amyloid Precursor Protein and triggers a proinflammatory phenotype in human monocytes53
Stu2 performs an essential kinetochore function independent of its microtubule polymerase activity53
ER-derived caveolin-coated vesicles transport newly synthesized cholesterol to the plasma membrane50
Fast-evolving cofactors regulate the role of HEATR5 complexes in intra-Golgi trafficking50
The phospholipids cardiolipin and phosphatidylethanolamine differentially regulate MDC biogenesis49
FGFR2 residence in primary cilia is necessary for epithelial cell signaling48
uPA-mediated remodeling of CCL21 gradients regulates lymphatic migration of dendritic cells47
Tubulin regulates stability and localization of STMN2 by binding preferentially to its soluble form47
A gene duplication of a septin reveals a developmentally regulated filament length control mechanism47
Migratory autolysosome disposal mitigates lysosome damage46
Taperin bundles F-actin at stereocilia pivot points enabling optimal lifelong mechanosensitivity46
Separating chromosomes, one way or another46
SARS-CoV-2 specific adaptations in N protein inhibit NF-κB activation and alter pathogenesis46
Organization of a cytoskeletal superstructure in the apical domain of intestinal tuft cells45
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